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10.1016/j.tins.2008.02.005 354

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In addition, we advocate the combination of medications and dietary modification for gouty patients in therapy, and caution that they should note the impact of nutritional factors on drug pharmacokinetics and pharmacodynamics

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Beta-3-AR knockout mice were completely unresponsive to chronic lipolytic effects, demonstrating that this receptor pathway is essential for sustained fat-reducing activity

The enhanced hepatic mitochondrial activity in the context of TKT deficiency has been shown to be the result of increased phospholipid synthesis, particularly increased synthesis of phosphatidylcholine which represents the major phospholipid in the mitochondrial membrane