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In patients, the ability to synthesize GSH in the lens decreases, and patients exhibit disrupted lens redox balance, reduced GSH concentrations, impaired GPX function, elevated iron redox levels, and increased lipid peroxidation, which indicates that age-related cataracts are closely related to ferroptosis (326, 327)

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To ensure synchronization among brain cell clocks, various mechanisms, including the release of melatonin by the pineal gland [171], come into play