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Despite this gap in knowledge, a plausible hypothesis could be formulated that either treatment with exogenous melatonin or interventions aimed at restoring endogenous melatonin secretion might offer potential in mitigating neurodegenerative processes induced by circadian disruption

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Transfusion-dependent -thalassemia patients may experience brain iron overload, potentially leading to cognitive impairment, in contrast to non-transfusion-dependent patients [43]

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