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In CKD patients, uremic toxins and ROS probably promote inflammation and oxidative stress through the stimulation of polymorphonuclear lymphocytes, leading to the release of inflammatory cytokines

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Multi-targeted therapeutic agents that impact both p53-dependent and independent checkpoints offer significant therapeutic potential, as these intersecting pathways ensure effective proliferation control while driving cancer cell death through apoptosis 46,47,48

Fivenson EM, Lautrup S, Sun N, Scheibye-Knudsen M, Stevnsner T, Nilsen H, et al