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medRxiv (preprint)

Evidence suggests that aberrant activation of lysosomal cathepsins, which play a key regulatory role in trypsinogen activation, may represent an early pathological event in acinar cell injury during the onset of AP

It is driven by iron-dependent phospholipid peroxidation and regulated by several cellular metabolic events, including redox homeostasis, iron handling, mitochondrial activity, and metabolism of amino acids, lipids and sugars [67]

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Theoretical Considerations BPC-157 is a strong promoter of new blood vessel growth