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The disease impairs fertility through multiple mechanisms, including anatomical distortions, a hostile pelvic environment due to chronic inflammation and oxidative stress, poor oocyte quality, impaired folliculogenesis, reduced ovarian reserve, and altered endometrial receptivity (6)

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It is reported that SA could reduce neuroinflammation, oxidative stress and cell apoptosis in cerebral ischemia-reperfusion injury, thereby ameliorating brain tissue damage, which may be involved in its inhibitory effect on excessive endoplasmic reticulum stress (54) indicated that SA could directly bind to the conserved cysteine residue 140 (Cys140) of IMPDH2, and further lead to IMPDH2 inactivation, thereby suppressing neuroinflammatory responses

PMID 16213110