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The mechanistic core of the finding was that Dihexa activated the PI3K/AKT signaling pathway in the brains of the APP/PS1 mice, and that the PI3K inhibitor wortmannin reversed Dihexas anti-inflammatory and anti-apoptotic effects [5]

Compared with vehicle-treated obese mice, the treated animals showed reductions in body-weight measures, white-adipose mass and adipocyte size

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It serves its intended purpose reliably in our daily lab activities

Available at: www.clinicalkey.com Accessed 6/9/2014