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beta-Amyloid-mediated vasoactivity and vascular endothelial damage

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However, notably, this protective effect could be disrupted by the antagonist of GHSR, [D-lys3]-GHRP-6 (116)

It is highly concentrated in skeletal muscle tissue and the heart, both of which utilize fatty acids as fuel

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Acting preventively either on the excessive production of free radicals in the noise-exposed cochlea and/or on noise-induced cochlear vasoconstriction, they may help to ease the consequences of NIHL