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(approximately 0.1%) ( 2 to generate significant amounts of O 2 -, the precursors to most ROS (Sohal, 1993)
This mechanism can be beneficial in injuries, as higher growth factor activity correlates with faster healing 3
and damaged type I alveolar epithelial cells are replaced by more ozone-resistant type II cells
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It also reduces muscle atrophy in diabetic models by alleviating mitochondrial dysfunction, endoplasmic reticulum stress, and apoptotic signaling (Liu et al., 2022)
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