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Al-Temimi, A

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With time microglia alter phenotype thus encouraging inflammation and neurodegeneration [40,43-45]

Furthermore, after acute UVB exposure, melanocytes in VDR-deficient mice exhibited reduced proliferative activity (PCNA/TYRP1), increased numbers of cells positive for the DNA damage marker CPD, and a significantly elevated apoptotic index (TUNEL/TYRP1), highlighting the protective role of VDR in mitigating UVB-induced genotoxic stress and sustaining melanocyte viability [134]

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Energy restriction and colorectal cancer: a call for additional research