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In summary, AT II infusion is sufficient to induce HDP and IL-6 production, and AT II inhibition is sufficient to prevent LPS-induced HDP, suggesting that the stimulation of AT II signaling underlies MIA and HDP
The uniform upregulation of important metabolites underscores a potential compensatory adaptation by the bacterium to maintain viability under the challenges of altered metabolic and oxidative states
Endothelial Cdk5 deficit leads to the development of spontaneous epilepsy through CXCL1/CXCR2-mediated reactive astrogliosis
(2023) stated that liver sinusoidal endothelial cells, being most vulnerable to damage, secrete damage-associated molecular patterns (DAMPs) dominated by HMGB1 to impact the whole liver microenvironment
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Physiol Plant 77:457464
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