Antimetastatic therapies of the polysulfide diallyl trisulfide against triple-negative breast cancer (TNBC) via suppressing MMP2/9 by blocking NF-kappaB and ERK/MAPK signaling pathways
Together, these observations suggest that BPC-157 functions as a modulator of interconnected signaling networks involving nitric oxide regulation, kinase mediated cytoskeletal signaling, and endothelial pathway activation in experimental biological systems
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