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However, more intensive studies are needed to gain a better understanding of the mechanisms of action ( The present study aimed to assess the potential therapeutic effects of PA on APAP-induced hepatotoxicity and the possible underlying mechanisms through its effects on CYP2E1 gene expression, serum and tissue iron load, oxidative stress and SIRT-1 expression

Interestingly, one month after the period of APAP intoxication, these mice showed increased glial burden without associated drivers, namely, bloodbrain barrier disruption, cholesterol accumulation, and elevation of inflammatory molecules in the brain and/or periphery

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Two case reports suggest that neonates and children have a reduced propensity to toxicity following paracetamol overdose due to differences in metabolism [50, 51]

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