HGF/c-Met signaling is a well-established oncogenic pathway: c-Met is amplified or overexpressed in multiple cancers (gastric, lung, renal, hepatocellular) HGF/c-Met activation promotes tumor cell proliferation, survival, invasion, and metastasis c-Met inhibitors are FDA-approved cancer drugs (cabozantinib, crizotinib, capmatinib) A compound that potentiates HGF/c-Met signaling raises theoretical concerns about promoting tumor growth or transformation with chronic use
Ignoring Hydration Both glutathione and collagen rely on adequate hydration to move through the body and reach the tissues where they are needed
What does reconstitution mean
cytosolic chaperon, methylmalonic aciduria, and homocystinuria type C protein FADH2
Combined with the patented BioMax-3 absorption system, its blood absorption rate is over 30% higher than similar products, with bioavailability approaching 95%
Jacobi MP