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Moreover, using conditional knockout mouse models (e.g., chondrocyte-specific Trpv1 / or Slc2a1 / ), the protective effects of CAT on ferroptosis and OA progression in vivo need to be verified, combined with spatial metabolomics to analyze dynamic changes in lipid peroxidation products and antioxidant molecules in joint tissues following CAT intervention

Role of Nicotinamide Adenine Dinucleotide and Related Precursors as Therapeutic Targets for Age-Related Degenerative Diseases: Rationale, Biochemistry, Pharmacokinetics, and Outcomes

The evidence base consists primarily of animal studies and anecdotal reports

Short-chain monounsaturated fatty acyl tails (MUFAs) replace long-chain PUFA tails in phospholipids because of the genetic deletion of ACSL4 [26, 28]

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