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Specifically, in vitro studies show a direct effect of GLP-1 on cardiomyocyte survival through activation of the RISK pathway in the presence of characteristic stimuli of ischemia-reperfusion injury and in the setting of HG (Table 1)

Therefore, although DPP-4 inhibitors have attractive clinical features, the benefits that might be expected from GLP-1 signaling may be undermined by their actions to enhance SDF-1

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[DOI] [PMC free article] [PubMed] [Google Scholar] 8.Choi H.K., Atkinson K., Karlson E.W., Willett W., Curhan G