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They do not constitute clinical evidence of efficacy or safety in humans, and all reference to biological activity pertains strictly to preclinical research settings

Some in vitro studies have shown that TNF may reduce melanocytestimulating hormone receptor binding activity, MC1R expression, or melanosomal protein gp87 expression, all of which promote melanogenesis

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PINK1 accumulates on the OMM in response to mitochondrial damage, recruiting Parkin to ubiquitinate mitochondrial proteins and promote autophagosome-mediated clearance [162]

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