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Under oxidative stress, the degradation of NRF2 by KEAP1 is activated [11]

However, the traditional application forms of PT 141, such as oral or injection, have problems such as low bioavailability, frequent administration, and significant side effects, which limit its widespread use

Future investigations should incorporate high-resolution joint-tissue single-cell transcriptomics, long-term OA models, and clinically stratified cohorts to further elucidate the diagnostic utility and mechanistic function of LPCAT3 in OA pathogenesis